IL-33 drives the antitumour effects of dendritic cells via upregulating CYLD expression in pulmonary adenocarcinoma-Reference-Cited by-同舟云学术

IL-33 drives the antitumour effects of dendritic cells via upregulating CYLD expression in pulmonary adenocarcinoma

Published:2019-04-09 Issue:1 Volume:47 Page:1335-1341
ISSN:2169-1401
Container-title:Artificial Cells, Nanomedicine, and Biotechnology
language:en
Short-container-title:Artificial Cells, Nanomedicine, and Biotechnology

Author:

Zhang Jiguang¹,Chen Yangming¹,Chen Kai¹,Huang Yunchao²,Xu Xunyu¹,Chen Qianshun¹,Huang Chen¹,Luo Jiewei³,Lin Xing¹

Affiliation:

1. Department of Thoracic Surgery, Fujian Provincial Hospital, Provincial Clinical College of Fujian Medical University, Fuzhou, Fujian, China;

2. Department of thoracic surgery, Yunnan Cancer Hospital, the Third Affiliated Hospital of Kunming Medical University, Kunming, Yunnan, China;

3. Department of traditional Chinese medicine, Fujian Province Hospital, School of clinical medicine, Fujian Medical University, Fuzhou, Fujian, China

Funder

Science and Technology Project of Fujian

The National Natural Science Fund

Publisher

Informa UK Limited

Subject

Pharmaceutical Science,Biomedical Engineering,General Medicine,Medicine (miscellaneous),Biotechnology

Link

https://www.tandfonline.com/doi/pdf/10.1080/21691401.2019.1596926

Reference43 articles.

1. Targeted BMI1 inhibition impairs tumor growth in lung adenocarcinomas with low CEBPα expression

2. Downregulation of Membrane Trafficking Proteins and Lactate Conditioning Determine Loss of Dendritic Cell Function in Lung Cancer

3. Interleukin 33 Signaling Restrains Sporadic Colon Cancer in an Interferon-γ–Dependent Manner

4. IL-33 promotes recovery from acute colitis by inducing miR-320 to stimulate epithelial restitution and repair

5. Interleukin-33 Receptor (ST2) Deficiency Improves the Outcome of Staphylococcus aureus-Induced Septic Arthritis

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