<i>Lef1</i> is transcriptionally activated by <i>Klf4</i> and suppresses hyperoxia-induced alveolar epithelial cell injury-Reference-Cited by-同舟云学术

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Lef1 is transcriptionally activated by Klf4 and suppresses hyperoxia-induced alveolar epithelial cell injury

Published:2022-08-11 Issue:7-8 Volume:48 Page:213-223
ISSN:0190-2148
Container-title:Experimental Lung Research
language:en
Short-container-title:Experimental Lung Research

Author:

Yang Min¹,Huang Xueshan²,Shen Fang³,Yi Juanjuan⁴,Meng Yanni¹,Chen Yanping¹

Affiliation:

1. Department of Respiratory, Hunan Children’s Hospital, Changsha, China

2. University of South China, Hengyang, China

3. Research Institute of Children, Hunan Children’s Hospital, Changsha, China

4. Department of Neonate, Hunan Children’s Hospital, Changsha, China

Funder

Youth Fund of Hunan Natural Science Foundation

Publisher

Informa UK Limited

Subject

Clinical Biochemistry,Pulmonary and Respiratory Medicine,Molecular Biology

Link

https://www.tandfonline.com/doi/pdf/10.1080/01902148.2022.2108945

Reference45 articles.

1. Decreased ZONAB expression promotes excessive transdifferentiation of alveolar epithelial cells in hyperoxia-induced bronchopulmonary dysplasia

2. Resveratrol alleviates alveolar epithelial cell injury induced by hyperoxia by reducing apoptosis and mitochondrial dysfunction

3. Role of the SENP1–SIRT1 pathway in hyperoxia-induced alveolar epithelial cell injury

4. Novel Strategies to Reduce Pulmonary Hypertension in Infants With Bronchopulmonary Dysplasia

5. Bronchopulmonary dysplasia: clinical aspects and preventive and therapeutic strategies

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