Coagulation F13A1 V34L, fibrinogen and homocysteine versus conventional risk factors in the pathogenesis of MI in young persons

Author:

Vishwajeet Vikarn1,Jamwal Manu2,Sharma Prashant2ORCID,Das Reena2ORCID,Ahluwalia Jasmina2,Dogra Rupinder Kaur2,Rohit Manoj Kumar3

Affiliation:

1. Department of Histopathology, Postgraduate Institute of Medical Education and Research, Chandigarh, India;

2. Department of Hematology, Postgraduate Institute of Medical Education and Research, Chandigarh, India;

3. Department of Cardiology, Postgraduate Institute of Medical Education and Research, Chandigarh, India

Publisher

Informa UK Limited

Subject

Cardiology and Cardiovascular Medicine,General Medicine

Reference38 articles.

1. World Health Organization Preventing Chronic Disease: A Vital Investment World Health Organization, Geneva 2005. Region and country specific information sheets: India; [Accessed on Oct 11, 2016]. Available from: http://www.who.int/chp/chronic_disease_report/media/india.pdf?ua=1

2. The factor XIII V34L polymorphism accelerates thrombin activation of factor XIII and affects cross-linked fibrin structure

3. Examining Thrombin Hydrolysis of the Factor XIII Activation Peptide Segment Leads to a Proposal for Explaining the Cardioprotective Effects Observed with the Factor XIII V34L Mutation

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