Chronic intermittent ethanol exposure-induced m6A modifications around mRNA stop codons of opioid receptor genes-Reference-Cited by-同舟云学术

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Chronic intermittent ethanol exposure-induced m6A modifications around mRNA stop codons of opioid receptor genes

Published:2023-12-20 Issue:1 Volume:19 Page:
ISSN:1559-2294
Container-title:Epigenetics
language:en
Short-container-title:Epigenetics

Author:

Liu Ying¹²,Koo Ji Sun³,Zhang Huiping¹²^ORCID

Affiliation:

1. Department of Psychiatry, Boston University Chobanian & Avedisian School of Medicine, Boston, USA

2. Department of Medicine (Biomedical Genetics), Boston University Chobanian & Avedisian School of Medicine, Boston, USA

3. Department of Biology, Boston University, Boston, USA

Funder

National Institute on Alcohol Abuse and Alcoholism

the 2022 Wing Tat Lee Award

Boston University

Publisher

Informa UK Limited

Subject

Cancer Research,Molecular Biology

Link

https://www.tandfonline.com/doi/pdf/10.1080/15592294.2023.2294515

Reference35 articles.

1. Genetic approaches to addiction: genes and alcohol

2. Identification of Methylated Nucleosides in Messenger RNA from Novikoff Hepatoma Cells

3. Comprehensive Analysis of mRNA Methylation Reveals Enrichment in 3′ UTRs and near Stop Codons

4. Topology of the human and mouse m6A RNA methylomes revealed by m6A-seq

5. m 6 A RNA Methylation Regulates the Self-Renewal and Tumorigenesis of Glioblastoma Stem Cells

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1. METTL14‐mediated upregulation of lncRNA HOTAIR represses PP1α expression by promoting H3K4me1 demethylation in oxycodone‐treated mice;CNS Neuroscience & Therapeutics;2024-07

2. Potential Molecular Mechanisms of Alcohol Use Disorder with Non-Coding RNAs and Gut Microbiota for the Development of Superior Therapeutic Application;Genes;2024-03-29

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