Sprouty4 is epigenetically upregulated in human colorectal cancer-Reference-Cited by-同舟云学术

Sprouty4 is epigenetically upregulated in human colorectal cancer

Published:2022-11-16 Issue:1 Volume:18 Page:
ISSN:1559-2294
Container-title:Epigenetics
language:en
Short-container-title:Epigenetics

Author:

Stuckel Alexei J.¹,Zeng Shuai²³,Lyu Zhen²³,Zhang Wei⁴,Zhang Xu⁵,Dougherty Urszula⁶,Mustafi Reba⁶,Khare Tripti¹,Zhang Qiong¹,Joshi Trupti²⁷⁸,Bissonnette Marc⁶,Khare Sharad¹⁹

Affiliation:

1. Department of Medicine, Division of Gastroenterology and Hepatology, University of Missouri, Columbia, Missouri, 65212, USA

2. Bond Life Sciences Center, University of Missouri, Columbia, Missouri, 65201, USA

3. Department of Electrical Engineering and Computer Science, University of Missouri, Columbia, Missouri, 65201, USA

4. Department of Preventive Medicine and the Robert H. Lurie Comprehensive Cancer Center, Northwestern University Feinberg School of Medicine, Chicago, Illinois, 60611, USA

5. Department of Medicine, University of Illinois, Chicago, Illinois, 60607, USA

6. Department of Medicine, Section of Gastroenterology, Hepatology and Nutrition; the University of Chicago, Chicago, Illinois, 60637, USA

7. Institute for Data Science and Informatics, University of Missouri, Columbia, Missouri, 65211, USA

8. Department of Health Management and Informatics; School of Medicine, University of Missouri, Columbia, Missouri, 65212, USA

9. Harry S. Truman Memorial Veterans’ Hospital, Columbia, Missouri, 65201, USA

Funder

U.S. Department of Veterans Affairs

Publisher

Informa UK Limited

Subject

Cancer Research,Molecular Biology

Link

https://www.tandfonline.com/doi/pdf/10.1080/15592294.2022.2145068

Reference48 articles.

1. Sprouty genes regulate proliferation and survival of human embryonic stem cells

2. IRF6 and SPRY4 Signaling Interact in Periderm Development

3. Sprouty3 and Sprouty4, Two Members of a Family Known to Inhibit FGF-Mediated Signaling, Exert Opposing Roles on Proliferation and Migration of Glioblastoma-Derived Cells

4. SPRY4-mediated ERK1/2 signaling inhibition abolishes 17β-estradiol-induced cell growth in endometrial adenocarcinoma cell

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