Rationale for IL-37 as a novel therapeutic agent in inflammation
Author:
Affiliation:
1. Ritchie Centre, Hudson Institute of Medical Research, Melbourne, Australia
2. Department of Paediatrics, School of Clinical Sciences, Monash University, Melbourne, Australia
3. Monash Newborn, Monash Children’s Hospital, Melbourne, Australia
Funder
National Health and Medical Research Council Investigator
Fielding Foundation’s Innovation
Publisher
Informa UK Limited
Subject
Immunology,Immunology and Allergy
Link
https://www.tandfonline.com/doi/pdf/10.1080/1744666X.2022.2108792
Reference31 articles.
1. IL-37 is a fundamental inhibitor of innate immunity
2. Suppression of antigen-specific adaptive immunity by IL-37 via induction of tolerogenic dendritic cells
3. Homodimerization attenuates the anti-inflammatory activity of interleukin-37
4. Monocytes and dendritic cells are the primary sources of interleukin 37 in human immune cells
5. IL-37 requires the receptors IL-18Rα and IL-1R8 (SIGIRR) to carry out its multifaceted anti-inflammatory program upon innate signal transduction
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