K+ deficiency caused defects in renal tubular cell proliferation, oxidative stress response, tissue repair and tight junction integrity, but enhanced energy production, proteasome function and cellular K+ uptake-Reference-Cited by-同舟云学术

K+ deficiency caused defects in renal tubular cell proliferation, oxidative stress response, tissue repair and tight junction integrity, but enhanced energy production, proteasome function and cellular K+ uptake

Published:2017-09-14 Issue:3 Volume:12 Page:247-258
ISSN:1933-6918
Container-title:Cell Adhesion & Migration
language:en
Short-container-title:Cell Adhesion & Migration

Author:

Kapincharanon Chompunoot¹²³,Thongboonkerd Visith¹³

Affiliation:

1. Medical Proteomics Unit, Office for Research and Development, Siriraj Hospital, Mahidol University, Bangkok, Thailand

2. Department of Immunology, Siriraj Hospital, Mahidol University, Bangkok, Thailand

3. Center for Research in Complex Systems Science, Mahidol University, Bangkok, Thailand

Publisher

Informa UK Limited

Subject

Cell Biology,Cellular and Molecular Neuroscience

Link

https://www.tandfonline.com/doi/pdf/10.1080/19336918.2017.1356554

Reference30 articles.

1. Pathophysiology and management of hypokalemia: a clinical perspective

2. Does hypokalemia contribute to acute kidney injury in chronic laxative abuse?

3. Proteomic identification of alterations in metabolic enzymes and signaling proteins in hypokalemic nephropathy

4. Chronic potassium depletion induces renal injury, salt sensitivity, and hypertension in young rats

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