High Hb A2β-Thalassemia Due to a 468 bp Deletion in a Patient with Hb S/β-Thalassemia
Author:
Publisher
Informa UK Limited
Subject
Biochemistry (medical),Clinical Biochemistry,Genetics (clinical),Hematology
Link
http://www.tandfonline.com/doi/pdf/10.1080/03630260500311651
Reference7 articles.
1. A New High A2‐β‐Thalassemia Due to a 468 bp Deletion (− 475 to − 8) in the β‐Globin Gene Promoter of the Intact β‐Globin Structural Gene
2. A beta-thalassemia gene caused by a 290-base pair deletion: analysis by direct sequencing of enzymatically amplified DNA
3. Observations on the levels of Hb A2 in patients with different beta- thalassemia mutations and a delta chain variant
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1. Characterization of Two Novel Deletions Involving the 5′ Region of the β-Globin Gene;Hemoglobin;2017-11-02
2. HbA2: biology, clinical relevance and a possible target for ameliorating sickle cell disease;British Journal of Haematology;2015-06-24
3. The genetics of hemoglobin A2regulation in sickle cell anemia;American Journal of Hematology;2014-08-04
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5. Other Sickle Hemoglobinopathies;Disorders of Hemoglobin
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