Inhalation of PM2.5 from diesel exhaust promote impairment of mitochondrial bioenergetics and dysregulate mitochondrial quality in rat heart: implications in isoproterenol-induced myocardial infarction model
Author:
Affiliation:
1. School of Chemical and Biotechnology, Vascular Biology lab, SASTRA Deemed University, Thanjavur, India
2. School of Chemical and Biotechnology, SASTRA Deemed University, Tirumalaisamudram, India
Publisher
Informa UK Limited
Subject
Health, Toxicology and Mutagenesis,Toxicology
Link
https://www.tandfonline.com/doi/pdf/10.1080/08958378.2022.2049931
Reference39 articles.
1. Hydrogen sulfide post-conditioning preserves interfibrillar mitochondria of rat heart during ischemia reperfusion injury
2. Label-free detection of uptake, accumulation, and translocation of diesel exhaust particles in ex vivo perfused human placenta
3. Exposure to diesel exhaust particulates induces cardiac dysfunction and remodeling
4. Effects of air pollution on mitochondrial function, mitochondrial DNA methylation, and mitochondrial peptide expression
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2. Consequential Impact of Particulate Matter Linked Inter-Fibrillar Mitochondrial Dysfunction in Rat Myocardium Subjected to Ischemia Reperfusion Injury;Biology;2022-12-13
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