Inhalation of PM<sub>2.5</sub> from diesel exhaust promote impairment of mitochondrial bioenergetics and dysregulate mitochondrial quality in rat heart: implications in isoproterenol-induced myocardial infarction model-Reference-Cited by-同舟云学术

Inhalation of PM_2.5 from diesel exhaust promote impairment of mitochondrial bioenergetics and dysregulate mitochondrial quality in rat heart: implications in isoproterenol-induced myocardial infarction model

Published:2022-03-15 Issue:5-6 Volume:34 Page:107-119
ISSN:0895-8378
Container-title:Inhalation Toxicology
language:en
Short-container-title:Inhalation Toxicology

Author:

Sivakumar Bhavana¹,Kurian Gino A.¹²

Affiliation:

1. School of Chemical and Biotechnology, Vascular Biology lab, SASTRA Deemed University, Thanjavur, India

2. School of Chemical and Biotechnology, SASTRA Deemed University, Tirumalaisamudram, India

Publisher

Informa UK Limited

Subject

Health, Toxicology and Mutagenesis,Toxicology

Link

https://www.tandfonline.com/doi/pdf/10.1080/08958378.2022.2049931

Reference39 articles.

1. Hydrogen sulfide post-conditioning preserves interfibrillar mitochondria of rat heart during ischemia reperfusion injury

2. Label-free detection of uptake, accumulation, and translocation of diesel exhaust particles in ex vivo perfused human placenta

3. Exposure to diesel exhaust particulates induces cardiac dysfunction and remodeling

4. Effects of air pollution on mitochondrial function, mitochondrial DNA methylation, and mitochondrial peptide expression

Cited by 5 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Comparative evaluation of single and multiple exposure to PM2.5 in respirable air on cardiac physiology, structure and function in a Wistar rat model;Journal of Environmental Sciences;2025-04

2. PM_2.5 toxicity in blood impairs cardiac redox balance and promotes mitochondrial dysfunction in rat heart that further aggravates ischemia reperfusion injury by modulating PI3K/AKT/mTOR/NF‐kB signaling axis;Journal of Biochemical and Molecular Toxicology;2024-05

3. Mitochondrial Dysfunction in Arrhythmia and Cardiac Hypertrophy;Reviews in Cardiovascular Medicine;2023-12-25

4. PM2.5-Induced Cardiac Structural Modifications and Declined Pro-Survival Signalling Pathways Are Responsible for the Inefficiency of GSK-3β Inhibitor in Attenuating Myocardial Ischemia-Reperfusion Injury in Rats;Cells;2023-08-15

5. Consequential Impact of Particulate Matter Linked Inter-Fibrillar Mitochondrial Dysfunction in Rat Myocardium Subjected to Ischemia Reperfusion Injury;Biology;2022-12-13