Brain injury, genotoxic damage and oxidative stress induced by Bromuconazole <i>in male Wistar rats</i> and in SH-SY5Y cell line-Reference-Cited by-同舟云学术

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Brain injury, genotoxic damage and oxidative stress induced by Bromuconazole in male Wistar rats and in SH-SY5Y cell line

Published:2022-06-20 Issue:6 Volume:27 Page:599-607
ISSN:1354-750X
Container-title:Biomarkers
language:en
Short-container-title:Biomarkers

Author:

Rjiba-Touati Karima¹²,Hamdi Hiba¹,M’nassri Asma¹,Rich Siwar¹,Mokni Moncef³,Abid Salwa¹

Affiliation:

1. Faculty of Dentistry, Laboratory of Research on Biologically Compatible Compounds, Monastir, Tunisia

2. Facutly of Science of Gafsa, University of Gafsa, Gafsa, Tunisia

3. Department of Anatomic Pathology and Histology, Hospital of Hached, Sousse, Tunisia

Funder

Ministry of Higher Education and Scientific Research

Publisher

Informa UK Limited

Subject

Health, Toxicology and Mutagenesis,Clinical Biochemistry,Biochemistry

Link

https://www.tandfonline.com/doi/pdf/10.1080/1354750X.2022.2087002

Reference45 articles.

1. Abdelhadya, D.H., El-Magd, M.A., Elbialy, Z.I., and Saleh, A.A. 2017. Bromuconazole-induced hepatotoxicity is accompanied by upregulation of P XR/CYP3A1 and downregulation of CAR/CYP2B1 gene expression. Toxicology mechanisms and methods, 27, 544–550.

2. A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

3. Protein Carbonyl Formation in Response to Propiconazole-Induced Oxidative Stress

4. Penconazole alters redox status, cholinergic function, and membrane-bound ATPases in the cerebrum and cerebellum of adult rats

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