Library-based lead compound discovery for CS-1 protein in multiple myeloma: homology modelling, molecular dynamic simulations, virtual screening and molecular docking-Reference-Cited by-同舟云学术

Library-based lead compound discovery for CS-1 protein in multiple myeloma: homology modelling, molecular dynamic simulations, virtual screening and molecular docking

Published:2021-08-30 Issue:16 Volume:47 Page:1358-1371
ISSN:0892-7022
Container-title:Molecular Simulation
language:en
Short-container-title:Molecular Simulation

Author:

Sepehrirad Shahrzad¹,Amanlou Arash²³^ORCID,Bagherzadeh Kowsar⁴⁵,Azizian Homa⁶,Amanlou Massoud³⁷^ORCID

Affiliation:

1. Tehran University of Medical Sciences, International campus (TUMS-IC), Tehran, Iran

2. Faculty of Specialized Veterinary Sciences, Science and Research Branch, Islamic Azad University, Tehran, Iran

3. Experimental Medicine Research Center, Tehran University of Medical Sciences, Tehran, Iran

4. Stem Cell and Regenerative Medicine Research Center, Iran University of Medical Sciences, Tehran, Iran

5. Eye Research Center, The Five Senses Institute, Rasoul Akram Hospital, Iran University of Medical Sciences, Tehran, Iran

6. Department of Medicinal Chemistry, School of Pharmacy, Iran University of Medical Sciences, Tehran, Iran

7. Department of Medicinal Chemistry, Faculty of Pharmacy, Tehran University of Medical Sciences, Tehran, Iran

Funder

Research Council of

Tehran University of Medical Sciences and Health Services

Publisher

Informa UK Limited

Subject

Condensed Matter Physics,General Materials Science,General Chemical Engineering,Modelling and Simulation,Information Systems,General Chemistry

Link

https://www.tandfonline.com/doi/pdf/10.1080/08927022.2021.1971225

Reference47 articles.

1. Multiple myeloma: 2020 update on diagnosis, risk‐stratification and management

2. CS1, a SLAM family receptor involved in immune regulation, is a therapeutic target in multiple myeloma

3. Multiple myeloma

4. Pathology and Epidemiology of Cancer

5. Molecular Cloning and Characterization of a Surface Antigen Preferentially Overexpressed on Multiple Myeloma Cells