Foot-and-mouth disease virus structural protein VP3 degrades Janus kinase 1 to inhibit IFN-γ signal transduction pathways-Reference-Cited by-同舟云学术

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Foot-and-mouth disease virus structural protein VP3 degrades Janus kinase 1 to inhibit IFN-γ signal transduction pathways

Published:2016-02-22 Issue:6 Volume:15 Page:850-860
ISSN:1538-4101
Container-title:Cell Cycle
language:en
Short-container-title:Cell Cycle

Author:

Li Dan,Wei Jin,Yang Fan,Liu Hua-Nan,Zhu Zi-Xiang,Cao Wei-Jun,Li Shu,Liu Xiang-Tao,Zheng Hai-Xue,Shu Hong-Bing

Publisher

Informa UK Limited

Subject

Cell Biology,Developmental Biology,Molecular Biology

Link

http://www.tandfonline.com/doi/pdf/10.1080/15384101.2016.1151584

Reference49 articles.

1. Foot-and-mouth disease under control in the UK

2. Distinctive features of foot-and-mouth disease virus, a member of the picornavirus family; aspects of virus protein synthesis, protein processing and structure

3. Cell mediated innate responses of cattle and swine are diverse during foot-and-mouth disease virus (FMDV) infection: A unique landscape of innate immunity

4. Immune evasion during foot-and-mouth disease virus infection of swine

5. Evading the host immune response: how foot-and-mouth disease virus has become an effective pathogen

Cited by 45 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Foot-and-mouth disease virus (FMDV) negatively regulates ZFP36 protein expression to alleviate its antiviral activity;Journal of Virology;2024-08-28

2. Encephalomyocarditis Virus Structural Protein VP3 Interacts with MAVS and Promotes its Autophagic Degradation to Interfere with the Type I Interferon Signaling Pathway;Frontiers in Bioscience-Landmark;2024-07-25

3. Picornavirus VP3 protein induces autophagy through the TP53-BAD-BAX axis to promote viral replication;Autophagy;2024-05-16

4. Foot-and-mouth disease virus structural protein VP3 interacts with HDAC8 and promotes its autophagic degradation to facilitate viral replication;Autophagy;2023-07-14

5. 3Cpro of FMDV inhibits type II interferon-stimulated JAK-STAT signaling pathway by blocking STAT1 nuclear translocation;Virologica Sinica;2023-06

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