Novel isatin-derived molecules activate p53 via interference with Mdm2 to promote apoptosis-Reference-Cited by-同舟云学术

Novel isatin-derived molecules activate p53 via interference with Mdm2 to promote apoptosis

Published:2018-08-03 Issue:15 Volume:17 Page:1917-1930
ISSN:1538-4101
Container-title:Cell Cycle
language:en
Short-container-title:Cell Cycle

Author:

Fedorova Olga¹,Daks Alexandra¹^ORCID,Petrova Varvara²,Petukhov Alexey¹³^ORCID,Lezina Larissa¹,Shuvalov Oleg¹,Davidovich Pavel⁴^ORCID,Kriger Darya¹,Lomert Ekaterina¹,Tentler Dmitry¹^ORCID,Kartsev Victor⁵,Uyanik Burhan⁶,Tribulovich Vyacheslav⁴,Demidov Oleg⁶,Melino Gerry²,Barlev Nickolai A.¹⁷^ORCID

Affiliation:

1. Gene Expression Programme, Institute of Cytology, Saint-Petersburg, Russia

2. MRC Toxicology Unit, Leicester, UK

3. Institute of Hematology, Almazov National Medical Research Centre, Russia

4. Molecular Pharmacology, State Technological University, Saint-Petersburg, Russia

5. Interbioscreen, Chernogolovka, Russia

6. INSERM U866, University of Burgundy, Dijon, France

7. Intracellular Signalling Laboratory, Moscow Institute of Physics and Technology, Dolgoprudny, Russia

Funder

Russian Foundation for Basic Research

Russian Science Foundation

Publisher

Informa UK Limited

Subject

Cell Biology,Developmental Biology,Molecular Biology

Link

https://www.tandfonline.com/doi/pdf/10.1080/15384101.2018.1506664

Reference53 articles.

1. Blinded by the Light: The Growing Complexity of p53

2. The microRNA and p53 families join forces against cancer

3. Transcriptional Regulation by P53

4. Expanding the p53 regulatory network: LncRNAs take up the challenge

5. miR-16 and miR-26a target checkpoint kinases Wee1 and Chk1 in response to p53 activation by genotoxic stress

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