Down-regulation of miR-20a-5p triggers cell apoptosis to facilitate mycobacterial clearance through targeting JNK2 in human macrophages-Reference-Cited by-同舟云学术

Down-regulation of miR-20a-5p triggers cell apoptosis to facilitate mycobacterial clearance through targeting JNK2 in human macrophages

Published:2016-08-11 Issue:18 Volume:15 Page:2527-2538
ISSN:1538-4101
Container-title:Cell Cycle
language:en
Short-container-title:Cell Cycle

Author:

Zhang Guoliang¹²,Liu Xi³,Wang Wenfei¹,Cai Yi¹,Li Shaoyuan¹,Chen Qi¹,Liao Mingfeng¹,Zhang Mingxia¹,Zeng Gucheng⁴,Zhou Boping¹,Feng Carl G.¹²,Chen Xinchun¹⁵

Affiliation:

1. Guangdong Key Lab for Diagnosis & Treatment of Emerging Infectious Diseases, Shenzhen Key Lab of Infection & Immunity, Shenzhen Third People's Hospital, Guangdong Medical University, Shenzhen, China

2. Department of Infectious Diseases and Immunology, Sydney Medical School, The University of Sydney, NSW, Australia

3. Department of Infectious Diseases, The Fifth Affiliated Hospital, Sun Yat-sen University, Zhuhai, China

4. Department of Microbiology, Zhongshan School of Medicine, Sun Yat-sen University, Guangzhou, China

5. Department of Pathogen, Shenzhen University School of Medicine, Shenzhen, China

Funder

Twelfth-Fifth Mega-Scientific Projects

Thirteenth-Fifth Mega-Scientific Projects

National Natural Science Foundation of China (NSFC)

Natural Science Foundation of Guangdong

Shenzhen Scientific and Technological Foundation

Publisher

Informa UK Limited

Subject

Cell Biology,Developmental Biology,Molecular Biology

Link

https://www.tandfonline.com/doi/pdf/10.1080/15384101.2016.1215386

Reference58 articles.

1. WHO. Global tuberculosis report 2015. 2015

2. Immunoevasion and immunosuppression of the macrophage byMycobacterium tuberculosis

3. Autophagy as an immune effector against tuberculosis

4. Apoptosis is an innate defense function of macrophages against Mycobacterium tuberculosis

5. A TNF- and c-Cbl-dependent FLIPS-degradation pathway and its function in Mycobacterium tuberculosis–induced macrophage apoptosis

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