Deficiency of XLF and PAXX prevents DNA double-strand break repair by non-homologous end joining in lymphocytes-Reference-Cited by-同舟云学术

Deficiency of XLF and PAXX prevents DNA double-strand break repair by non-homologous end joining in lymphocytes

Published:2017-01-19 Issue:3 Volume:16 Page:286-295
ISSN:1538-4101
Container-title:Cell Cycle
language:en
Short-container-title:Cell Cycle

Author:

Hung Putzer J.¹²,Chen Bo-Ruei¹²,George Rosmy²,Liberman Caleb²,Morales Abigail J.¹²,Colon-Ortiz Pedro¹,Tyler Jessica K.¹,Sleckman Barry P.¹²,Bredemeyer Andrea L.²

Affiliation:

1. Department of Pathology and Laboratory Medicine, Weill Cornell Medical College, New York, NY, USA

2. Department of Pathology and Immunology, Washington University School of Medicine, St. Louis, MO, USA

Publisher

Informa UK Limited

Subject

Cell Biology,Developmental Biology,Molecular Biology

Link

https://www.tandfonline.com/doi/pdf/10.1080/15384101.2016.1253640

Reference50 articles.

1. The Mechanism of Double-Strand DNA Break Repair by the Nonhomologous DNA End-Joining Pathway

2. The DNA Damage Response: Making It Safe to Play with Knives

3. Functional overlaps between XLF and the ATM-dependent DNA double strand break response

4. Modernizing the Nonhomologous End-Joining Repertoire: Alternative and Classical NHEJ Share the Stage

5. Somatic generation of antibody diversity

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