Low-dose methotrexate enhances cycling of highly anaplastic cancer cells
Author:
Affiliation:
1. Department of Experimental and Clinical Biomedical Sciences “Mario Serio,” Università degli Studi di Firenze, Florence, Italy
Publisher
Informa UK Limited
Subject
Cell Biology,Developmental Biology,Molecular Biology
Link
https://www.tandfonline.com/doi/pdf/10.1080/15384101.2016.1252883
Reference22 articles.
1. The involvement of a Nanog, Klf4 and c-Myc transcriptional circuitry in the intertwining between neoplastic progression and reprogramming
2. Hypoxia-resistant profile implies vulnerability of cancer stem cells to physiological agents, which suggests new therapeutic targets
3. Resistance Mechanisms to Methotrexate in Tumors
4. The respiration-linked limiting step of tumor cell transition from the non-cycling to the cycling state: Its inhibition by oxidizable substrates and its relationships to purine metabolism
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