DNA methylation of the Oct4A enhancers in embryonal carcinoma cells after etoposide treatment is associated with alternative splicing and altered pluripotency in reversibly senescent cells-Reference-Cited by-同舟云学术

DNA methylation of the Oct4A enhancers in embryonal carcinoma cells after etoposide treatment is associated with alternative splicing and altered pluripotency in reversibly senescent cells

Published:2018-02-01 Issue:3 Volume:17 Page:362-366
ISSN:1538-4101
Container-title:Cell Cycle
language:en
Short-container-title:Cell Cycle

Author:

Baryshev Mikhail¹,Inashkina Inna²,Salmina Kristine²,Huna Anda²^ORCID,Jackson Thomas R³^ORCID,Erenpreisa Jekaterina²^ORCID

Affiliation:

1. August Kirchenstein Institute of Microbiology and Virology, Riga Stradins University, Ratsupites 5, LV-1067 Riga, Latvia

2. Latvian Biomedical Research & Study Centre, Ratsupites 1, Riga LV-1067, Latvia

3. Faculty Institute for Cancer Sciences, University of Manchester, Manchester, M13 9WL, UK

Funder

Europe Social Fund Project

Publisher

Informa UK Limited

Subject

Cell Biology,Developmental Biology,Molecular Biology

Link

https://www.tandfonline.com/doi/pdf/10.1080/15384101.2018.1426412

Reference28 articles.

1. Accelerated Senescence of Cancer Stem Cells: A Failure to Thrive or a Route to Survival?

2. Embryonic stem (ES) cells and embryonal carcinoma (EC) cells: opposite sides of the same coin

3. Role of stress-activated OCT4A in the cell fate decisions of embryonal carcinoma cells treated with etoposide

4. DNA damage causes TP53-dependent coupling of self-renewal and senescence pathways in embryonal carcinoma cells

5. Core Transcriptional Regulatory Circuitry in Human Embryonic Stem Cells

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