Human CTC1 promotes TopBP1 stability and CHK1 phosphorylation in response to telomere dysfunction and global replication stress-Reference-Cited by-同舟云学术

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Human CTC1 promotes TopBP1 stability and CHK1 phosphorylation in response to telomere dysfunction and global replication stress

Published:2020-12-03 Issue:24 Volume:19 Page:3491-3507
ISSN:1538-4101
Container-title:Cell Cycle
language:en
Short-container-title:Cell Cycle

Author:

Ackerson Stephanie M.¹,Gable Caroline I.¹,Stewart Jason A.¹

Affiliation:

1. Department of Biological Sciences, University of South Carolina, Columbia, SC, USA

Funder

the National Institutes of Health

National Institute of General Medical Sciences

University of South Carolina

Publisher

Informa UK Limited

Subject

Cell Biology,Developmental Biology,Molecular Biology

Link

https://www.tandfonline.com/doi/pdf/10.1080/15384101.2020.1849979

Reference66 articles.

1. ATM, ATR, and DNA-PK: The Trinity at the Heart of the DNA Damage Response

2. The essential kinase ATR: ensuring faithful duplication of a challenging genome

3. ATR disruption leads to chromosomal fragmentation and early embryonic lethality

4. Targeted disruption of the cell-cycle checkpoint gene ATR leads to early embryonic lethality in mice

5. Functional uncoupling of MCM helicase and DNA polymerase activities activates the ATR-dependent checkpoint

Cited by 14 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Human CST Stimulates Base Excision Repair to Prevent the Accumulation of Oxidative DNA Damage;Journal of Molecular Biology;2024-08

2. Suppression of CTC1 inhibits hepatocellular carcinoma cell growth and enhances RHPS4 cytotoxicity;Molecular Biology Reports;2024-07-13

3. Guardians of the Genome: How the Single-Stranded DNA-Binding Proteins RPA and CST Facilitate Telomere Replication;Biomolecules;2024-02-22

4. Human CST complex restricts excessive PrimPol repriming upon UV induced replication stress by suppressing p21;Nucleic Acids Research;2024-02-13

5. Poly(ADP-Ribose) Polymerase-1 Lacking Enzymatic Activity Is Not Compatible with Mouse Development;Cells;2023-08-16

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