FEM1 proteins are ancient regulators of SLBP degradation-Reference-Cited by-同舟云学术

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FEM1 proteins are ancient regulators of SLBP degradation

Published:2017-02-13 Issue:6 Volume:16 Page:556-564
ISSN:1538-4101
Container-title:Cell Cycle
language:en
Short-container-title:Cell Cycle

Author:

Dankert John F.¹²,Pagan Julia K.¹²³,Starostina Natalia G.⁴,Kipreos Edward T.⁴,Pagano Michele¹²³

Affiliation:

1. Department of Biochemistry and Molecular Pharmacology, New York University, School of Medicine, New York, NY, USA

2. Perlmutter NYU Cancer Center, New York University, School of Medicine, New York, NY, USA

3. Howard Hughes Medical Institute, New York University School of Medicine, New York, NY, USA

4. Department of Cellular Biology, University of Georgia, Athens, GA, USA

Publisher

Informa UK Limited

Subject

Cell Biology,Developmental Biology,Molecular Biology

Link

https://www.tandfonline.com/doi/pdf/10.1080/15384101.2017.1284715

Reference38 articles.

1. Building and remodelling Cullin–RING E3 ubiquitin ligases

2. VHL-box and SOCS-box domains determine binding specificity for Cul2-Rbx1 and Cul5-Rbx2 modules of ubiquitin ligases

3. Mechanisms and function of substrate recruitment by F-box proteins

4. A CUL-2 Ubiquitin Ligase Containing Three FEM Proteins Degrades TRA-1 to Regulate C. elegans Sex Determination

5. A sex-determining gene, fem-1, required for both male and hermaphrodite development in Caenorhabditis elegans

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1. Hominini-specific regulation of the cell cycle by stop codon readthrough of FEM1B;Journal of Cell Science;2024-08-15

2. Structural insights into the ubiquitylation strategy of the oligomeric CRL2FEM1B E3 ubiquitin ligase;The EMBO Journal;2024-02-15

3. A novel de novo FEM1C variant is linked to neurodevelopmental disorder with absent speech, pyramidal signs and limb ataxia;Human Molecular Genetics;2022-11-07

4. A novelde novoFEM1C variant is linked to neurodevelopmental disorder with absent speech, pyramidal signs, and limb ataxia;2022-04-24

5. Human cytomegalovirus lytic infection inhibits replication-dependent histone synthesis and requires stem loop binding protein function;Proceedings of the National Academy of Sciences;2022-03-28

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