p53 and Mdm2 act synergistically to maintain cardiac homeostasis and mediate cardiomyocyte cell cycle arrest through a network of microRNAs-Reference-Cited by-同舟云学术

p53 and Mdm2 act synergistically to maintain cardiac homeostasis and mediate cardiomyocyte cell cycle arrest through a network of microRNAs

Published:2017-08-11 Issue:17 Volume:16 Page:1585-1600
ISSN:1538-4101
Container-title:Cell Cycle
language:en
Short-container-title:Cell Cycle

Author:

Stanley-Hasnain Shanna¹²,Hauck Ludger²,Grothe Daniela²,Aschar-Sobbi Roozbeh³,Beca Sanja³,Butany Jagdish⁴,Backx Peter H.³⁵⁶,Mak Tak W.⁷,Billia Filio¹²³⁶⁸^ORCID

Affiliation:

1. Institute of Medical Science, University of Toronto, Toronto, Ontario, Canada

2. Toronto General Research Institute, Toronto, Ontario, Canada

3. Department of Physiology, University of Toronto, Toronto, Ontario, Canada

4. Department of Pathology, University Health Network, Toronto, Ontario, Canada

5. Department of Biology, York University, Toronto, Ontario, Canada

6. Heart and Stroke Richard Lewar Centre of Excellence, University of Toronto, Toronto, Ontario, Canada

7. Campbell Family Cancer Research Institute, Princess Margaret Hospital, Toronto, Ontario, Canada

8. Division of Cardiology and Peter Munk Cardiac Centre, University Health Network, Toronto, Ontario, Canada

Publisher

Informa UK Limited

Subject

Cell Biology,Developmental Biology,Molecular Biology

Link

https://www.tandfonline.com/doi/pdf/10.1080/15384101.2017.1346758

Reference73 articles.

1. Unravelling mechanisms of p53-mediated tumour suppression

2. Stress-Dependent Nucleolin Mobilization Mediated by p53-Nucleolin Complex Formation

3. p53 Requires the Stress Sensor USF1 to Direct Appropriate Cell Fate Decision

4. Homeostatic functions of the p53 tumor suppressor: Regulation of energy metabolism and antioxidant defense

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