Δ133p53: A p53 isoform enriched in human pluripotent stem cells
Author:
Affiliation:
1. Laboratory of Human Carcinogenesis, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD, USA
Publisher
Informa UK Limited
Subject
Cell Biology,Developmental Biology,Molecular Biology
Link
https://www.tandfonline.com/doi/pdf/10.1080/15384101.2017.1345228
Reference7 articles.
1. Autophagic degradation of the inhibitory p53 isoform Δ133p53α as a regulatory mechanism for p53-mediated senescence
2. p53 isoforms regulate aging- and tumor-associated replicative senescence in T lymphocytes
3. p53 isoforms regulate astrocyte-mediated neuroprotection and neurodegeneration
4. Δ133p53 represses p53-inducible senescence genes and enhances the generation of human induced pluripotent stem cells
5. p53 isoform Δ133p53 promotes efficiency of induced pluripotent stem cells and ensures genomic integrity during reprogramming
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