Cell senescence, rapamycin and hyperfunction theory of aging
Author:
Affiliation:
1. Roswell Park Comprehensive Cancer Center, Buffalo, NY, USA
Funder
reported that there is no funding associated with the work featured in this article
Publisher
Informa UK Limited
Subject
Cell Biology,Developmental Biology,Molecular Biology
Link
https://www.tandfonline.com/doi/pdf/10.1080/15384101.2022.2054636
Reference72 articles.
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3. Premature senescence involving p53 and p16 is activated in response to constitutive MEK/MAPK mitogenic signaling
4. The mitogen-activated protein kinase pathway mediates growth arrest or E1A-dependent apoptosis in SKBR3 human breast cancer cells
5. Tumor suppressor activity of the ERK/MAPK pathway by promoting selective protein degradation
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