A DDB2 mutant protein unable to interact with PCNA promotes cell cycle progression of human transformed embryonic kidney cells
Author:
Affiliation:
1. Dipartimento di Medicina Molecolare; Unità di Immunologia e Patologia generale; Università di Pavia; Pavia, Italy
2. Istituto di Genetica Molecolare (IGM) del CNR; Pavia, Italy
Publisher
Informa UK Limited
Subject
Cell Biology,Developmental Biology,Molecular Biology
Link
https://www.tandfonline.com/doi/pdf/10.1080/15384101.2015.1120921
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3. Malignant melanoma cells acquire resistance to DNA interstrand cross-linking chemotherapeutics by p53-triggered upregulation of DDB2/XPC-mediated DNA repair
4. A method to measure the duration of DNA syntheses and the potential doubling time from a single sample
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1. Epithelial-to-mesenchymal transition and NF-kB pathways are promoted by a mutant form of DDB2, unable to bind PCNA, in UV-damaged human cells;BMC Cancer;2024-05-21
2. Exploring new potential role of DDB2 by host cell reactivation assay in human tumorigenic cells;BMC Cancer;2019-10-29
3. Emerging Roles of DDB2 in Cancer;International Journal of Molecular Sciences;2019-10-18
4. A damaged DNA binding protein 2 mutation disrupting interaction with proliferating-cell nuclear antigen affects DNA repair and confers proliferation advantage;Biochimica et Biophysica Acta (BBA) - Molecular Cell Research;2018-06
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