<i>NPM1</i> and <i>DNMT3A</i> mutations are associated with distinct blast immunophenotype in acute myeloid leukemia-Reference-Cited by-同舟云学术

NPM1 and DNMT3A mutations are associated with distinct blast immunophenotype in acute myeloid leukemia

Published:2022-05-06 Issue:1 Volume:11 Page:
ISSN:2162-402X
Container-title:OncoImmunology
language:en
Short-container-title:OncoImmunology

Author:

Kuželová Kateřina¹^ORCID,Brodská Barbora¹^ORCID,Marková Jana²,Petráčková Martina³,Schetelig Johannes⁴,Ransdorfová Šárka⁵,Gašová Zdenka⁶⁷,Šálek Cyril²⁷

Affiliation:

1. Department of Proteomics, Institute of Hematology and Blood Transfusion, Prague, Czech Republic

2. Clinical Department, Institute of Hematology and Blood Transfusion, Prague, Czech Republic

3. Department of Gene Immunotherapy Research, Institute of Hematology and Blood Transfusion, Prague, Czech Republic

4. Medical Clinic I, Division Hematology, Cell Therapy, and Medical Oncology, University Hospital Carl Gustav Carus, Technical University Dresden, Dresden, Germany

5. Department of Cytogenetics, Institute of Hematology and Blood Transfusion, Prague, Czech Republic

6. Department of Apheresis, Institute of Hematology and Blood Transfusion, Prague, Czech Republic

7. Institute of Clinical and Experimental Hematology, 1st Faculty of Medicine, Charles University, Prague, Czech Republic

Funder

European Regional Development Fund

Czech Republic

Ministry of Health of the Czech Republic

Publisher

Informa UK Limited

Subject

Oncology,Immunology,Immunology and Allergy

Link

https://www.tandfonline.com/doi/pdf/10.1080/2162402X.2022.2073050

Reference58 articles.

1. Microenvironment Produced by Acute Myeloid Leukemia Cells Prevents T Cell Activation and Proliferation by Inhibition of NF-κB, c-Myc, and pRb Pathways

2. Signatures of CD8+ T cell dysfunction in AML patients and their reversibility with response to chemotherapy

3. Peripheral blood T cells in acute myeloid leukemia (AML) patients at diagnosis have abnormal phenotype and genotype and form defective immune synapses with AML blasts

4. Reversible suppression of T cell function in the bone marrow microenvironment of acute myeloid leukemia

5. Increased PD-1+Tim-3+ exhausted T cells in bone marrow may influence the clinical outcome of patients with AML

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