Crk adaptor protein promotes PD-L1 expression, EMT and immune evasion in a murine model of triple-negative breast cancer-Reference-Cited by-同舟云学术

Crk adaptor protein promotes PD-L1 expression, EMT and immune evasion in a murine model of triple-negative breast cancer

Published:2017-09-27 Issue:1 Volume:7 Page:e1376155
ISSN:2162-402X
Container-title:OncoImmunology
language:en
Short-container-title:OncoImmunology

Author:

Kumar Sushil¹,Davra Viralkumar¹,E. Obr Alison²,Geng Ke¹,L. Wood Teresa²,De Lorenzo Mariana S.³,Birge Raymond B.¹

Affiliation:

1. Department of Microbiology, Biochemistry and Molecular Genetics, Cancer Center, Rutgers- New Jersey Medical School, 205 South Orange Ave, Newark, NJ, USA

2. Department of Pharmacology, Physiology and Neuroscience, Rutgers University, Cancer Center, Rutgers- New Jersey Medical School, 205 South Orange Ave, Newark, NJ, USA

3. Department of Cell Biology & Molecular Medicine, Rutgers- New Jersey Medical School, Newark, NJ, USA

Publisher

Informa UK Limited

Subject

Oncology,Immunology,Immunology and Allergy

Link

https://www.tandfonline.com/doi/pdf/10.1080/2162402X.2017.1376155

Reference41 articles.

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2. v-Crk activates the phosphoinositide 3-kinase/AKT pathway in transformation

3. Identification and characterization of a high-affinity interaction between v-Crk and tyrosine-phosphorylated paxillin in CT10-transformed fibroblasts.

4. A 31-amino-acid N-terminal extension regulates c-Crk binding to tyrosine-phosphorylated proteins.

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