AIM2 fosters lung adenocarcinoma immune escape by modulating PD-L1 expression in tumor-associated macrophages via JAK/STAT3-Reference-Cited by-同舟云学术

AIM2 fosters lung adenocarcinoma immune escape by modulating PD-L1 expression in tumor-associated macrophages via JAK/STAT3

Published:2023-10-25 Issue:3 Volume:19 Page:
ISSN:2164-5515
Container-title:Human Vaccines & Immunotherapeutics
language:en
Short-container-title:Human Vaccines & Immunotherapeutics

Author:

Ye Hua¹,Yu Wenwen¹,Li Yunlei¹,Bao Xiaoqiong¹,Ni Yangyang¹,Chen Xiangxiang¹,Sun Yangjie¹,Chen Ali¹,Zhou Weilong¹,Li Jifa¹

Affiliation:

1. Department of Respiratory and Critical Care Medicine of Affiliated Yueqing Hospital, Wenzhou Medical University, Wenzhou, China

Funder

no funding

Publisher

Informa UK Limited

Subject

Pharmacology,Immunology,Immunology and Allergy

Link

https://www.tandfonline.com/doi/pdf/10.1080/21645515.2023.2269790

Reference37 articles.

1. circRNA-002178 act as a ceRNA to promote PDL1/PD1 expression in lung adenocarcinoma

2. Landscape and dynamics of single tumor and immune cells in early and advanced‐stage lung adenocarcinoma

3. Infiltration of CD8(+) FOXP3(+) T cells, CD8(+) T cells, and FOXP3(+) T cells in non-small cell lung cancer microenvironment;Hao J;Int J Clin Exp Pathol,2020

4. Progranulin induces immune escape in breast cancer via up-regulating PD-L1 expression on tumor-associated macrophages (TAMs) and promoting CD8+ T cell exclusion

5. MiRNA-Mediated Macrophage Polarization and its Potential Role in the Regulation of Inflammatory Response

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