HMGB1 is increased in adolescents with polycystic ovary syndrome (PCOS) and decreases after treatment with myo-inositol (MYO) in combination with alpha-lipoic acid (ALA)
Author:
Affiliation:
1. Department of Obstetrics Gynaecology and Paediatrics, Azienda USL-IRCCS di Reggio Emilia, Reggio Emilia, Italy;
2. Department of Obstetrics and Gynaecology, University of Cagliari, Cagliari, Italy
Publisher
Informa UK Limited
Subject
Obstetrics and Gynecology,Endocrinology,Endocrinology, Diabetes and Metabolism
Link
https://www.tandfonline.com/doi/pdf/10.1080/09513590.2020.1725967
Reference64 articles.
1. Criteria, prevalence, and phenotypes of polycystic ovary syndrome
2. Criteria for Defining Polycystic Ovary Syndrome as a Predominantly Hyperandrogenic Syndrome: An Androgen Excess Society Guideline
3. The follicular excess in polycystic ovaries, due to intra-ovarian hyperandrogenism, may be the main culprit for the follicular arrest
4. New perspectives on the definition and management of polycystic ovary syndrome
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1. Biomarker potential of competing endogenous RNA networks in Polycystic Ovary Syndrome (PCOS);Non-coding RNA Research;2024-06
2. Myoinositol or D-chiro-inositol for PCOS symptoms in adolescents: a narrative review;Journal of Pediatric Endocrinology and Metabolism;2023-12-21
3. Signaling pathways and targeted therapeutic strategies for polycystic ovary syndrome;Frontiers in Endocrinology;2023-10-19
4. Emodin improves glucose metabolism and ovarian function in PCOS mice via the HMGB1/TLR4/NF-κB molecular pathway;Reproduction;2023-08-31
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