Catestatin enhances ATP-induced activation of glial cells mediated by purinergic receptor P2X4-Reference-Cited by-同舟云学术

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Catestatin enhances ATP-induced activation of glial cells mediated by purinergic receptor P2X4

Published:2021-01-27 Issue: Volume: Page:1-9
ISSN:1079-9893
Container-title:Journal of Receptors and Signal Transduction
language:en
Short-container-title:Journal of Receptors and Signal Transduction

Author:

Du Errong¹,Wang Anhui¹,Fan Rongping²,Rong Lilou²,Yang Runan¹,Xing Juping¹,Shi Xiangchao³,Qiao Bao³,Yu Ruoyang³,Xu Changshui¹⁴

Affiliation:

1. Department of Physiology, Basic Medical College of Nanchang University, Nanchang, P.R. China

2. The Fourth Clinical Medical College of Nanchang University, Nanchang, P.R. China

3. Queen Mary School, Medical Department, Nanchang University–Queen Mary University of London, Nanchang, P.R. China

4. Jiangxi Provincial Key Laboratory of Autonomic Nervous Function and Disease, Nanchang, P.R. China

Funder

National Natural Science Foundation of China

Innovation Fund of the Graduate School of Nanchang University

Publisher

Informa UK Limited

Subject

Cell Biology,Molecular Biology,Biochemistry

Link

https://www.tandfonline.com/doi/pdf/10.1080/10799893.2021.1878536

Reference27 articles.

1. Gap junction mediated signaling between satellite glia and neurons in trigeminal ganglia

2. The contribution of satellite glial cells to chemotherapy-induced neuropathic pain

3. Altered functional properties of satellite glial cells in compressed spinal ganglia

4. Pathological and protective roles of glia in chronic pain

5. Chapter 22 Contribution of glia to pain processing in health and disease

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Naringin inhibits P2X4 receptor expression on satellite glial cells in the neonatal rat dorsal root ganglion;Journal of Applied Biomedicine;2023-12-18

2. Gallic Acid Improves Comorbid Chronic Pain and Depression Behaviors by Inhibiting P2X7 Receptor-Mediated Ferroptosis in the Spinal Cord of Rats;ACS Chemical Neuroscience;2023-01-31

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