Modifications in GPR21 and GPR82 genes expression as a consequence of metabolic syndrome etiology-Reference-Cited by-同舟云学术

Modifications in GPR21 and GPR82 genes expression as a consequence of metabolic syndrome etiology

Published:2020-06-25 Issue:1 Volume:41 Page:38-44
ISSN:1079-9893
Container-title:Journal of Receptors and Signal Transduction
language:en
Short-container-title:Journal of Receptors and Signal Transduction

Author:

Romero-Nava Rodrigo¹²,García Noemí³,Aguayo-Cerón Karla Aidee¹,Sánchez Muñoz Fausto⁴,Huang Fengyang²,Hong Enrique⁵,Villafaña Santiago¹

Affiliation:

1. Laboratorio de Señalización Intracelular, Sección de Estudios de Posgrado, Escuela Superior de Medicina del Instituto Politécnico Nacional, Ciudad de México, México

2. Laboratorio de Investigación en Farmacología, Hospital Infantil de México Federico Gómez (HIMFG), Ciudad de México, México

3. Escuela Nacional de Medicina, Tecnológico de Monterrey, Monterrey, México

4. Departamento de Inmunología, Instituto Nacional de Cardiología Ignacio Chávez, Ciudad de México, México

5. Departamento de Neurofarmacobiología, Centro de Investigación y de Estudios Avanzados, Ciudad de México, México

Publisher

Informa UK Limited

Subject

Cell Biology,Molecular Biology,Biochemistry

Link

https://www.tandfonline.com/doi/pdf/10.1080/10799893.2020.1784228

Reference45 articles.

1. Miglitol, α-glycosidase inhibitor, reduces visceral fat accumulation and cardiovascular risk factors in subjects with the metabolic syndrome: A randomized comparable study

2. Harmonizing the Metabolic Syndrome

3. Obésité, diabète de type 2 et perturbateurs endocriniens

4. Allopurinol alleviates hypertension and proteinuria in high fructose, high salt and high fat induced model of metabolic syndrome

5. Low cellular IRS 1 gene and protein expression predict insulin resistance and NIDDM

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