Montelukast ameliorates doxorubicin-induced cardiotoxicity via modulation of p-glycoprotein and inhibition of ROS-mediated TNF-α/NF-κB pathways-Reference-Cited by-同舟云学术

Montelukast ameliorates doxorubicin-induced cardiotoxicity via modulation of p-glycoprotein and inhibition of ROS-mediated TNF-α/NF-κB pathways

Published:2020-02-27 Issue: Volume: Page:1-12
ISSN:0148-0545
Container-title:Drug and Chemical Toxicology
language:en
Short-container-title:Drug and Chemical Toxicology

Author:

Hafez Heba M.¹,Hassanein Hanaa²

Affiliation:

1. Department of Pharmacology, Faculty of Medicine, Minia University, Minia, Egypt;

2. Department of Histology, Faculty of Medicine, Minia University, Minia, Egypt

Publisher

Informa UK Limited

Subject

Chemical Health and Safety,Health, Toxicology and Mutagenesis,Public Health, Environmental and Occupational Health,Pharmacology,Toxicology,General Medicine

Link

https://www.tandfonline.com/doi/pdf/10.1080/01480545.2020.1730885

Reference63 articles.

1. Mechanisms underlying the protective effect of montelukast in prevention of endometrial hyperplasia in female rats

2. Vitamin C mitigates oxidative/nitrosative stress and inflammation in doxorubicin-induced cardiomyopathy

3. Doxorubicin-induced oxidative stress: The protective effect of nicorandil on HL-1 cardiomyocytes

4. Genistein protects against doxorubicin‐induced cardiotoxicity through Nrf‐2/HO‐1 signaling in mice model

5. Inhibition of leukotriene C4 action reduces oxidative stress and apoptosis in cardiomyocytes and impedes remodeling after myocardial injury

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