Molecular mechanisms linking geranylgeranyl diphosphate synthase to cell survival and proliferation
Author:
Affiliation:
1. Department of Pharmaceutical Sciences, University of Connecticut, Storrs, CT, USA;
2. Institute for Systems Genomics, University of Connecticut, Storrs, CT, USA
Funder
Funding was provided in part by the National Institutes of Health under Award Number
Publisher
Informa UK Limited
Subject
Cell Biology,Molecular Biology
Link
https://www.tandfonline.com/doi/pdf/10.1080/09687688.2016.1213432
Reference94 articles.
1. p21 WAF1/CIP1 Is Upregulated by the Geranylgeranyltransferase I Inhibitor GGTI-298 through a Transforming Growth Factor β- and Sp1-Responsive Element: Involvement of the Small GTPase RhoA
2. Mutationally activated Rho GTPases in cancer
3. RhoA Prenylation Is Required for Promotion of Cell Growth and Transformation and Cytoskeleton Organization but Not for Induction of Serum Response Element Transcription
4. Farnesylated RhoB Prevents Cell Cycle Arrest and Actin Cytoskeleton Disruption Caused by the Geranylgeranyltransferase I Inhibitor GGTI-298
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