Xanthine oxidoreductase knockout mice with high HPRT activity were not rescued by NAD+replenishment-Reference-Cited by-同舟云学术

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Xanthine oxidoreductase knockout mice with high HPRT activity were not rescued by NAD+replenishment

Published:2020-03-04 Issue:10-12 Volume:39 Page:1465-1473
ISSN:1525-7770
Container-title:Nucleosides, Nucleotides & Nucleic Acids
language:en
Short-container-title:Nucleosides, Nucleotides & Nucleic Acids

Author:

Hosoyamada Makoto¹^ORCID,Tomioka Naoko H.¹,Ohtsubo Toshio²,Ichida Kimiyoshi³

Affiliation:

1. Department of Human Physiology and Pathology, Faculty of Pharma-Science, Teikyo University, Tokyo, Japan;

2. Department of Internal Medicine (Hypertension), Fukuoka Red Cross Hospital, Fukuoka, Japan;

3. Department of Pathophysiology, Tokyo University of Pharmacy and Life Sciences, Tokyo, Japan

Publisher

Informa UK Limited

Subject

Genetics,Molecular Medicine,Biochemistry,General Medicine

Link

https://www.tandfonline.com/doi/pdf/10.1080/15257770.2020.1725044

Reference12 articles.

1. Hyperuricemia and urate nephropathy in urate oxidase-deficient mice.

2. Xanthine Oxidoreductase Is an Endogenous Regulator of Cyclooxygenase-2

3. Production of a model for Lesch–Nyhan syndrome in hypoxanthine phosphoribosyltransferase–deficient mice

4. Urat1-Uox double knockout mice are experimental animal models of renal hypouricemia and exercise-induced acute kidney injury

5. Activity of purine phosphoribosyltransferases and of two enzymes of pyrimidine biosynthesis in erythrocytes of ten mammalian species

Cited by 3 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Kidney Failure Secondary to Hereditary Xanthinuria due to a Homozygous Deletion of the XDH Gene in the Absence of Overt Kidney Stone Disease;Nephron;2024

2. Lack of xanthine dehydrogenase leads to a remarkable renal decline in a novel hypouricemic rat model;iScience;2022-09

3. SLC23A3 is a renal hypoxanthine transporter;Nucleosides, Nucleotides & Nucleic Acids;2022-01-30

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