Glutathione Trisulfide Prevents Lipopolysaccharide-induced Inflammatory Gene Expression in Retinal Pigment Epithelial Cells-Reference-Cited by-同舟云学术

Glutathione Trisulfide Prevents Lipopolysaccharide-induced Inflammatory Gene Expression in Retinal Pigment Epithelial Cells

Published:2020-11-20 Issue: Volume: Page:1-12
ISSN:0927-3948
Container-title:Ocular Immunology and Inflammation
language:en
Short-container-title:Ocular Immunology and Inflammation

Author:

Tawarayama Hiroshi¹²^ORCID,Suzuki Noriyuki¹,Inoue-Yanagimachi Maki¹,Himori Noriko¹,Tsuda Satoru¹,Sato Kota¹³,Ida Tomoaki⁴,Akaike Takaaki⁴,Kunikata Hiroshi¹²,Nakazawa Toru¹²³⁵⁶

Affiliation:

1. Department of Ophthalmology, Tohoku University Graduate School of Medicine, Sendai, Japan

2. Department of Retinal Disease Control, Tohoku University Graduate School of Medicine, Sendai, Japan

3. Department of Ophthalmic Imaging and Information Analytics, Tohoku University Graduate School of Medicine, Sendai, Japan

4. Department of Environmental Medicine and Molecular Toxicology, Tohoku University Graduate School of Medicine, Sendai, Japan

5. Collaborative Program of Ophthalmic Drug Discovery, Tohoku University Graduate School of Medicine, Sendai, Japan

6. Department of Advanced Ophthalmic Medicine, Tohoku University Graduate School of Medicine, Sendai, Japan

Funder

Japan Agency for Medical Research and Development

Publisher

Informa UK Limited

Subject

Ophthalmology,Immunology and Allergy

Link

https://www.tandfonline.com/doi/pdf/10.1080/09273948.2020.1833224

Reference68 articles.

1. Retinal Pigment Epithelium-immune System Interactions: Cytokine Production and Cytokine-induced Changes

2. Comparison of the effects of recombinant interleukin 6 and recombinant interleukin 1 on nonspecific resistance to infection

3. Synergistic effects of gamma interferon on inflammatory mediators that induce interleukin-6 gene expression and secretion by human retinal pigment epithelial cells

4. Quercetin Inhibits the Production of IL-1β-Induced Inflammatory Cytokines and Chemokines in ARPE-19 Cells via the MAPK and NF-κB Signaling Pathways

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