Silencing of ALOX15 reduces ferroptosis and inflammation induced by cerebral ischemia-reperfusion by regulating PHD2/HIF2α signaling pathway
Author:
Affiliation:
1. Department of Cerebrovascular Disease, People’s Hospital of Leshan, Leshan City, Sichuan provincial, China
Funder
Research Project Plan
Publisher
Informa UK Limited
Subject
Molecular Biology,Bioengineering,Biotechnology
Link
https://www.tandfonline.com/doi/pdf/10.1080/02648725.2023.2210449
Reference31 articles.
1. Hypoxia exacerbates nonalcoholic fatty liver disease via the HIF-2α/PPARα pathway
2. Ferritin reduction is essential for cerebral ischemia-induced hippocampal neuronal death through p53/SLC7A11-mediated ferroptosis
3. Introduction for Focused Updates in Cerebrovascular Disease
4. Endothelial Conditional Knockdown of NMMHC IIA (Nonmuscle Myosin Heavy Chain IIA) Attenuates Blood-Brain Barrier Damage During Ischemia-Reperfusion Injury
5. Ferroptosis: mechanisms, biology and role in disease
Cited by 2 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Rhythm gene PER1 mediates ferroptosis and lipid metabolism through SREBF2/ALOX15 axis in polycystic ovary syndrome;Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease;2024-06
2. Deciphering Ferroptosis: From Molecular Pathways to Machine Learning-Guided Therapeutic Innovation;Molecular Biotechnology;2024-04-13
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