Genetic Mapping of Brain Plasticity Across Development in Williams Syndrome: ERP Markers of Face and Language Processing-Reference-Cited by-同舟云学术

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Genetic Mapping of Brain Plasticity Across Development in Williams Syndrome: ERP Markers of Face and Language Processing

Published:2013-11 Issue:8 Volume:38 Page:613-642
ISSN:8756-5641
Container-title:Developmental Neuropsychology
language:en
Short-container-title:Developmental Neuropsychology

Author:

Mills D. L.,Dai L.,Fishman I.,Yam A.,Appelbaum L. G.,St. George M.,Galaburda A.,Bellugi U.,Korenberg J. R.

Publisher

Informa UK Limited

Subject

Developmental and Educational Psychology,Neuropsychology and Physiological Psychology

Link

http://www.tandfonline.com/doi/pdf/10.1080/87565641.2013.825617

Reference69 articles.

1. A cross-syndrome study of the development of holistic face recognition in children with autism, Down syndrome, and Williams syndrome

2. Partial 7q11.23 deletions further implicate GTF2I and GTF2IRD1 as the main genes responsible for the Williams-Beuren syndrome neurocognitive profile

3. Dorsal-stream motion processing deficits persist into adulthood in Williams syndrome

4. I. The Neurocognitive Profile of Williams Syndrome: A Complex Pattern of Strengths and Weaknesses

5. Bridging cognition, the brain and molecular genetics: evidence from Williams syndrome

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1. Neuropsychological Genotype–Phenotype in Patients with Williams Syndrome with Atypical Deletions: A Systematic Review;Neuropsychology Review;2022-12-15

2. A single‐session behavioral protocol for successful event‐related potential recording in children with neurodevelopmental disorders;Developmental Psychobiology;2021-09-14

3. An EEG investigation of alpha and beta activity during resting states in adults with Williams syndrome;BMC Psychology;2021-05-05

4. Abnormal auditory event-related potentials in Williams syndrome;European Journal of Medical Genetics;2021-03

5. Core transcriptional networks in Williams syndrome: IGF1-PI3K-AKT-mTOR, MAPK and actin signaling at the synapse echo autism;Human Molecular Genetics;2021-02-09

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