Pasireotide treatment does not modify hyperglycemic and corticosterone acute restraint stress responses in rats-Reference-Cited by-同舟云学术

Pasireotide treatment does not modify hyperglycemic and corticosterone acute restraint stress responses in rats

Published:2018-04-17 Issue:4 Volume:21 Page:370-375
ISSN:1025-3890
Container-title:Stress
language:en
Short-container-title:Stress

Author:

Ribeiro-Oliveira Antônio¹,Schweizer Junia R. O. L¹,Amaral Pedro H. S.¹,Bizzi Mariana F.¹,Silveira Warley Cezar da¹,Espirito-Santo Daniel T. A.¹,Zille Giancarlo¹,Soares Beatriz S.¹,Schmid Herbert A.²,Yuen Kevin C. J.³

Affiliation:

1. Department of Internal Medicine, Laboratory of Endocrinology, School of Medicine, Federal University of Minas Gerais, Belo Horizonte, MG, Brazil;

2. Department of Oncology Research, Novartis Institutes for BioMedical Research, Novartis Pharma AG, Basel, Switzerland;

3. Department of Neuroendocrinology, Barrow Pituitary Center, Barrow Neurological Institute, Phoenix, AZ, USA

Funder

CNPq

FAPEMIG

Publisher

Informa UK Limited

Subject

Behavioral Neuroscience,Psychiatry and Mental health,Endocrine and Autonomic Systems,Neuropsychology and Physiological Psychology,Physiology

Link

https://www.tandfonline.com/doi/pdf/10.1080/10253890.2018.1451838

Reference28 articles.

1. Management of hyperglycemia associated with pasireotide (SOM230): Healthy volunteer study

2. Paraventricular nucleus modulates autonomic and neuroendocrine responses to acute restraint stress in rats

3. Angiotensin-converting enzyme inhibition changes the metabolic response to neuroglucopenic stress

4. Effect of SOM230 (Pasireotide) on Corticotropic Cells: Action in Dogs with Cushing’s Disease

5. Agonist-Biased Signaling at the sst2A Receptor: The Multi-Somatostatin Analogs KE108 and SOM230 Activate and Antagonize Distinct Signaling Pathways

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