Loss of PFKFB4 induces cell cycle arrest and glucose metabolism inhibition by inactivating MEK/ERK/c-Myc pathway in cervical cancer cells
Author:
Affiliation:
1. Gynecology, Three Gorges Hospital Affiliated to Chongqing University, Chongqing, PR China
2. Obstetrics, Three Gorges Hospital Affiliated to Chongqing University, Chongqing, PR China
Publisher
Informa UK Limited
Subject
Obstetrics and Gynecology
Link
https://www.tandfonline.com/doi/pdf/10.1080/01443615.2022.2062225
Reference27 articles.
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3. Identification and validation of a six-gene signature associated with glycolysis to predict the prognosis of patients with cervical cancer
4. MicroRNA-451 induces epithelial–mesenchymal transition in docetaxel-resistant lung adenocarcinoma cells by targeting proto-oncogene c-Myc
5. Cancer Cell Metabolism: There Is No ROS for the Weary: Figure 1.
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1. A novel prognostic biomarker in progression free survival for patients with cervical cancer, glucose to c-reactive protein ratio (GCR);BMC Cancer;2024-05-23
2. Comparative clinical significance and biological roles of PFKFB family members in oral squamous cell carcinoma;Cancer Cell International;2023-11-02
3. THOC3 interacts with YBX1 to promote lung squamous cell carcinoma progression through PFKFB4 mRNA modification;Cell Death & Disease;2023-07-27
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