Interleukin (IL)-33 immunobiology in asthma and airway inflammatory diseases
Author:
Affiliation:
1. Department of Internal Medicine, Division of Allergy and Immunology, University of Nebraska Medical Center, Omaha, NE, USA
Funder
National Institute of Environmental Health Sciences
National Institute for Occupational Safety and Health
Central States Center of Agricultural Safety and Health.
Publisher
Informa UK Limited
Subject
Pulmonary and Respiratory Medicine,Immunology and Allergy,Pediatrics, Perinatology and Child Health
Link
https://www.tandfonline.com/doi/pdf/10.1080/02770903.2021.2020815
Reference72 articles.
1. Interleukin-33 (IL-33): A nuclear cytokine from the IL-1 family
2. Nuclear IL-33 restrains the early conversion of fibroblasts to an extracellular matrix-secreting phenotype
3. The R213G polymorphism in SOD3 protects against allergic airway inflammation
4. In vitro Culture with Cytokines Provides a Tool to Assess the Effector Functions of ILC2s in Peripheral Blood in Asthma
5. MyD88 regulates a prolonged adaptation response to environmental dust exposure-induced lung disease
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2. Expression of pro-inflammatory cytokines (IL-18, IL-33) at the level of the mucous membrane, the entry point of infection, in persons who have experienced COVID-19 disease;Russian Journal of Infection and Immunity;2024-07-28
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4. Endoplasmic reticulum stress drives macrophages to produce IL-33 to favor Th2 polarization in the airways;Journal of Leukocyte Biology;2024-03-22
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