Control of the Aggressive Capacity of Prostate Cancer by Nutritional Inhibitors of Urokinase and Lipoxygenase
Author:
Affiliation:
1. Urology Research Center, Department of Urology, Medical College of Ohio [J.J., S. H. S., E.S-J.], Physiology and Molecular Medicine, [J.J., S.H.S.], 3065 Arlington Ave., Toledo, OH 43614-5807, USA Telephone: 419-383-3691, Fax: 419-383-3168
Publisher
Kamla Raj Enterprises
Subject
Genetics (clinical),Genetics
Link
https://www.tandfonline.com/doi/pdf/10.1080/09723757.2003.11885838
Reference11 articles.
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2. Hart CA, Scott LJ, Bagley S, Bryden AA, Clarke NW, Lang SH 2002. Role of proteolytic enzymes in human prostate bone metastasis formation: in vivo and in vitro studies.Br J Cancer,86(7): 1136–1142.
3. Koeneman KS, Yeung F, Chung LW 1999. Osteomimetic properties of prostate cancer cells: A hypothesis supporting the predilection of prostate cancer metastasis and growth in the bone environment.Prostate,39(4): 246–261.
4. Kumar S, Ziereis K, Wiegrebe W, Muller K 2000. Medicinal plants from nepal: evaluation as inhibitors of leukotriene biosynthesis.J Ethnopharmacol,70(3): 191–195.
5. Onesti S, Matthews DJ, Aducci P, Amiconi Bolognesi M,Menegatti E, Ascenzi P 1992. Binding of the Kunitz-type trypsin inhibitor DE-3 from Erythrina caffra seeds to serine proteinases: A comparative study.J Mol Recognit,5(3): 105–114.
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