The ameliorative effects of Nigella sativa, thymoquinone, and bentonite against aflatoxicosis in broilers via AFAR and Nrf2 signalling pathways, and down-regulation of caspase-3-Reference-Cited by-同舟云学术

The ameliorative effects of Nigella sativa, thymoquinone, and bentonite against aflatoxicosis in broilers via AFAR and Nrf2 signalling pathways, and down-regulation of caspase-3

Published:2021-12-06 Issue: Volume: Page:1-8
ISSN:0007-1668
Container-title:British Poultry Science
language:en
Short-container-title:British Poultry Science

Author:

Ates M. B.¹^ORCID,Ortatatli M.¹^ORCID,Oguz H.²^ORCID,Ozdemir O.¹^ORCID,Terzi F.³^ORCID,Ciftci M. K.⁴^ORCID,Hatipoglu F.¹⁵^ORCID

Affiliation:

1. Faculty of Veterinary Medicine, Department of Pathology, Selcuk University, Konya, Turkey

2. Faculty of Veterinary Medicine, Department of Pharmacology and Toxicology, Selcuk University, Konya, Turkey

3. Faculty of Veterinary Medicine, Department of Pathology, Kastamonu University, Kastamonu, Turkey

4. Faculty of Dentistry, Department of Basic Sciences, Istanbul Gelisim University, Istanbul, Turkey

5. Faculty of Veterinary Medicine, Department of Pathology, Kyrgyz-Turkish Manas University, Bishkek, Kyrgyzstan

Funder

The Scientific and Technological Research Council of Turkey

Selcuk University Scientific Research Projects Coordination Office

Publisher

Informa UK Limited

Subject

Animal Science and Zoology,General Medicine,Food Science

Link

https://www.tandfonline.com/doi/pdf/10.1080/00071668.2021.1998366

Reference50 articles.

1. Pharmacological and toxicological properties of Nigella sativa

2. The effects of Nigella sativa seeds and thymoquinone on aflatoxin phase-2 detoxification through glutathione and glutathione-S-transferase alpha-3, and the relationship between aflatoxin B1-DNA adducts in broilers

3. Phase-1 bioactivation mechanisms of aflatoxin through AhR, CAR and PXR nuclear receptors and the interactions with Nigella sativa seeds and thymoquinone in broilers

4. Thymoquinone alleviates nonalcoholic fatty liver disease in rats via suppression of oxidative stress, inflammation, apoptosis

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