Regulation of T cell differentiation by the AP-1 transcription factor JunB
Author:
Affiliation:
1. Department of Biochemistry, Toho University School of Medicine, Tokyo, Japan
2. Faculty of Medicine, Division of Rheumatology, Department of Internal Medicine, Ohashi Medical Center, Toho University, Tokyo, Japan
Publisher
Informa UK Limited
Subject
Immunology,Immunology and Allergy
Link
https://www.tandfonline.com/doi/pdf/10.1080/25785826.2021.1872838
Reference77 articles.
1. Different members of the jun proto-oncogene family exhibit distinct patterns of expression in response to type beta transforming growth factor.
2. AP-1 subunits: quarrel and harmony among siblings
3. JunB is essential for mammalian placentation
4. Mice lacking JunB are osteopenic due to cell-autonomous osteoblast and osteoclast defects
5. Psoriasis-like skin disease and arthritis caused by inducible epidermal deletion of Jun proteins
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