An antibody to IP-10 is a potent antagonist of cell migrationin vitroandin vivoand does not affect disease in several animal models of inflammation-Reference-Cited by-同舟云学术

An antibody to IP-10 is a potent antagonist of cell migrationin vitroandin vivoand does not affect disease in several animal models of inflammation

Published:2009-01 Issue:3 Volume:42 Page:171-182
ISSN:0891-6934
Container-title:Autoimmunity
language:en
Short-container-title:Autoimmunity

Author:

Byrne Fergus R.,Winters Aaron,Brankow David,Hu Sylvia,Juan Todd,Steavenson Shirley,Doellgast George,Byrne Fergus R.,Winters Aaron,Brankow David,Hu Sylvia,Juan Todd,Steavenson Shirley,Doellgast George,Kuchimanchi Kamesh,Brown Heather,Anderson Sharon,Smelt Sara,Sullivan Tim,Alcorn Dina,Tocker Joel,Dean Charley,Macmaster John,Kirchner Jacqueline,Buys Janet,Manoukian Raffi,Jiao Eric,Zou Xiaoming,Campanella Gabriele S.,Siu Gerald

Publisher

Informa UK Limited

Subject

Immunology,Immunology and Allergy

Link

http://www.tandfonline.com/doi/pdf/10.1080/08916930802629547

Reference48 articles.

1. Oligomerization of CXCL10 Is Necessary for Endothelial Cell Presentation and In Vivo Activity

2. Human eosinophils produce the T cell-attracting chemokines MIG and IP-10 upon stimulation with IFN-γ

3. Constitutive and regulated expression and secretion of interferon-γ-inducible protein 10 (IP-10/CXCL10) in human adipocytes

4. Substance P Enhances the Production of Interferon-induced Protein of 10 kDa by Human Keratinocytes in Synergy with Interferon-γ

5. TNF-α IS A POTENT INDUCER FOR IFN-INDUCIBLE PROTEIN-10 IN HEPATOCYTES AND UNAFFECTED BY GM-CSF IN VIVO, IN CONTRAST TO IL-1β AND IFN-γ

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