Zanubrutinib for the treatment of Waldenström Macroglobulinemia
Author:
Affiliation:
1. Department of Haematology, St Vincent’s Hospital, Melbourne, Australia
2. Department of Haematology, Peter MacCallum Cancer Centre, Melbourne, Australia
3. Department of Medicine, University of Melbourne, Melbourne, Australia
Publisher
Informa UK Limited
Subject
Hematology
Link
https://www.tandfonline.com/doi/pdf/10.1080/17474086.2020.1851184
Reference52 articles.
1. MYD88 L265P Somatic Mutation in Waldenström's Macroglobulinemia
2. A mutation in MYD88 (L265P) supports the survival of lymphoplasmacytic cells by activation of Bruton tyrosine kinase in Waldenström macroglobulinemia
3. The genomic landscape of Waldenström macroglobulinemia is characterized by highly recurring MYD88 and WHIM-like CXCR4 mutations, and small somatic deletions associated with B-cell lymphomagenesis
4. The WHIM-like CXCR4S338X somatic mutation activates AKT and ERK, and promotes resistance to ibrutinib and other agents used in the treatment of Waldenstrom’s Macroglobulinemia
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1. Zanubrutinib Ameliorates Cardiac Fibrosis and Inflammation Induced by Chronic Sympathetic Activation;Molecules;2023-08-12
2. Zanubrutinib attenuates bleomycin-induced pulmonary fibrosis by inhibiting the TGF-β1 signaling pathway;International Immunopharmacology;2022-12
3. Coming of Age for BTK Inhibitor Therapy: A Review of Zanubrutinib in Waldenström Macroglobulinemia;Cells;2022-10-19
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