Combination treatment of docosahexaenoic acid (DHA) and all-trans-retinoic acid (ATRA) inhibit IL-17 and RORγt gene expression in PBMCs of patients with relapsing-remitting multiple sclerosis-Reference-Cited by-同舟云学术

Combination treatment of docosahexaenoic acid (DHA) and all-trans-retinoic acid (ATRA) inhibit IL-17 and RORγt gene expression in PBMCs of patients with relapsing-remitting multiple sclerosis

Published:2017-11-18 Issue:1 Volume:40 Page:11-17
ISSN:0161-6412
Container-title:Neurological Research
language:en
Short-container-title:Neurological Research

Author:

Mousavi Nasl-khameneh Ateke¹,Mirshafiey Abbas²,Naser Moghadasi Abdorreza³,Chahardoli Reza⁴,Mahmoudi Maryam⁵,Parastouei Karim⁵,Yekaninejad Mir Saeed⁶^ORCID,Saboor-Yaraghi Ali Akbar²⁵

Affiliation:

1. Department of Cellular and Molecular Nutrition, School of Nutritional Sciences and Dietetics, International Campus, Tehran University of Medical Sciences, Tehran, Iran

2. Department of Immunology, School of Public Health, Tehran University of Medical Sciences, Tehran, Iran

3. Department of Neurology and MS Research Center, Neuroscience Institute, Sina Hospital, Tehran University of Medical Sciences, Tehran, Iran

4. Endocrine Research Center, Research Institute for Endocrine Sciences, Shahid Beheshti University of Medical Sciences, Tehran, Iran

5. Department of Cellular and Molecular Nutrition, School of Nutritional Sciences and Dietetics, Tehran University of Medical Sciences, Tehran, Iran

6. Department of Epidemiology and Biostatistics, School of Public Health, Tehran University of Medical Sciences, Tehran, Iran

Funder

TUMS

Publisher

Informa UK Limited

Subject

Clinical Neurology,Neurology,General Medicine

Link

https://www.tandfonline.com/doi/pdf/10.1080/01616412.2017.1382800

Reference43 articles.

1. CD4+T-cell subsets in inflammatory diseases: beyond the Th1/Th2 paradigm

2. A distinct lineage of CD4 T cells regulates tissue inflammation by producing interleukin 17

3. Phenotypical and functional characterization of T helper 17 cells in multiple sclerosis

4. Th17 and regulatory T cell balance in autoimmune and inflammatory diseases

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