Marked biochemical difference in amyloid proportion between intra- and extraocular tissues in a liver-transplanted patient with hereditary ATTR amyloidosis-Reference-Cited by-同舟云学术

Marked biochemical difference in amyloid proportion between intra- and extraocular tissues in a liver-transplanted patient with hereditary ATTR amyloidosis

Published:2017-01-02 Issue:1 Volume:24 Page:17-23
ISSN:1350-6129
Container-title:Amyloid
language:en
Short-container-title:Amyloid

Author:

Yoshinaga Tsuneaki¹,Yazaki Masahide¹²,Kametani Fuyuki³,Sekijima Yoshiki¹²,Iesato Yasuhiro⁴,Miyahara Teruyoshi⁴,Tsuchiya-Suzuki Ayako¹,Sano Kenji⁵,Higuchi Keiichi²⁶,Ikeda Shu-ichi¹

Affiliation:

1. Department of Medicine (Neurology and Rheumatology), Shinshu University School of Medicine, Matsumoto, Japan,

2. Department of Biological Sciences for Intractable Neurological Diseases, Institute for Biomedical Sciences, Shinshu University, Matsumoto, Japan,

3. Department of Dementia and Higher Brain Function, Tokyo Metropolitan Institute of Medical Science, Tokyo, Japan,

4. Department of Ophthalmology, Shinshu University School of Medicine, Matsumoto, Japan,

5. Department of Laboratory Medicine, Shinshu University School of Medicine, Matsumoto, Japan, and

6. Department of Aging Biology, Institute of Pathogenesis and Disease Prevention, Shinshu University Graduate School of Medicine, Mastumoto, Japan

Publisher

Informa UK Limited

Subject

Internal Medicine

Link

https://www.tandfonline.com/doi/pdf/10.1080/13506129.2016.1276055

Reference25 articles.

1. Familial transthyretin-type amyloid polyneuropathy in Japan: Clinical and genetic heterogeneity

2. Biochemical effect of liver transplantation in two Swedish patients with familial amyloidotic polyneuropathy (FAP-met30)

3. Cardiac Amyloid in Patients with Familial Amyloid Polyneuropathy Consists of Abundant Wild-Type Transthyretin

4. Progressive Wild-Type Transthyretin Deposition after Liver Transplantation Preferentially Occurs onto Myocardium in FAP Patients

5. DE NOVO AMYLOID SYNTHESIS IN OCULAR TISSUE IN FAMILIAL AMYLOIDOTIC POLYNEUROPATHY AFTER LIVER TRANSPLANTATION

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