Protecting effect of emodin in experimental autoimmune encephalomyelitis mice by inhibiting microglia activation and inflammation via Myd88/PI3K/Akt/NF-κB signalling pathway-Reference-Cited by-同舟云学术

Protecting effect of emodin in experimental autoimmune encephalomyelitis mice by inhibiting microglia activation and inflammation via Myd88/PI3K/Akt/NF-κB signalling pathway

Published:2022-04-01 Issue:4 Volume:13 Page:9322-9344
ISSN:2165-5979
Container-title:Bioengineered
language:en
Short-container-title:Bioengineered

Author:

Zheng Kenan¹²,Lv Baojiang¹²,Wu Lulu¹²,Wang Chen³,Xu Haoyou⁴,Li Xiaojun⁵,Wu Zhibing⁶,Zhao Yuanqi⁴,Zheng Zequan⁴⁷^ORCID

Affiliation:

1. The First Clinical School, Guangzhou University of Chinese Medicine, Guangzhou, China

2. Lingnan Medical Research Center, Guangzhou University of Chinese Medicine, Guangzhou, China

3. Department of Traditional Chinese Medicine, Zhujiang Hospital, Southern Medical University, Guangzhou, China

4. Department of Neurology, The Second Affiliated Hospital of Guangzhou University of Chinese Medicine, Guangdong Provincial Hospital of Traditional Chinese Medicine, Guangzhou, China

5. The Second Clinical School, Guangzhou University of Chinese Medicine, Guangzhou, China

6. Department of Neurology, The First Affiliated Hospital of Guangzhou University of Chinese Medicine, Guangzhou, China

7. Doctor of equivalent degree, Guangzhou University of Chinese Medicine, Guangzhou, China

Funder

Natural Science Foundation of Guangdong Province

the Guangdong Provincial Hospital of traditional Chinese Medicine Science and Technology research project

Traditional Chinese Medicine Bureau of Guangdong Province

Publisher

Informa UK Limited

Subject

Applied Microbiology and Biotechnology,General Medicine,Bioengineering,Biotechnology

Link

https://www.tandfonline.com/doi/pdf/10.1080/21655979.2022.2052671

Reference72 articles.

1. The changing course of multiple sclerosis: rising incidence, change in geographic distribution, disease course, and prognosis

2. Microglial autophagy–associated phagocytosis is essential for recovery from neuroinflammation

3. Multiple sclerosis: experimental models and reality

4. Pien Tze Huang Alleviates Relapsing-Remitting Experimental Autoimmune Encephalomyelitis Mice by Regulating Th1 and Th17 Cells

5. Neurons promote encephalitogenic CD4+ lymphocyte infiltration in experimental autoimmune encephalomyelitis

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