Hyperoxia induces alveolar epithelial cell apoptosis by regulating mitochondrial function through small mothers against decapentaplegic 3 (SMAD3) and extracellular signal-regulated kinase 1/2 (ERK1/2)
Author:
Affiliation:
1. Department of Pediatric Intensive Care Unit, Guizhou Provincial People’s Hospital, Guiyang, China
2. Department of Pediatrics, Affiliated Hospital of Hebei University, Baoding, China
Funder
National Natural Science Foundation of China
Publisher
Informa UK Limited
Subject
Applied Microbiology and Biotechnology,General Medicine,Bioengineering,Biotechnology
Link
https://www.tandfonline.com/doi/pdf/10.1080/21655979.2021.2012953
Reference35 articles.
1. Long term consequences of oxygen therapy in the neonatal period
2. Bronchopulmonary Dysplasia: Chronic Lung Disease of Infancy and Long-Term Pulmonary Outcomes
3. Hyperoxia and Apoptosis in Developing Mouse Lung Mesenchyme
4. Control Mechanisms of Lung Alveolar Development and Their Disorders in Bronchopulmonary Dysplasia
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