Prostaglandin E1-Induced Hyperostosis: Clinicopathologic Correlations and Possible Pathogenetic Mechanisms-Reference-Cited by-同舟云学术

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Prostaglandin E1-Induced Hyperostosis: Clinicopathologic Correlations and Possible Pathogenetic Mechanisms

Published:1996-01 Issue:3 Volume:16 Page:489-507
ISSN:1077-1042
Container-title:Pediatric Pathology & Laboratory Medicine
language:en
Short-container-title:Pediatric Pathology & Laboratory Medicine

Author:

Faye-Petersen Ona M.,Johnson Walter H.,Carlo Waldemar A.,Hedlund Gary L.,Pacifico Albert D.,Blair Harry C.

Publisher

Informa UK Limited

Subject

Pathology and Forensic Medicine,Pediatrics, Perinatology and Child Health

Link

http://www.tandfonline.com/doi/pdf/10.1080/15513819609168686

Reference31 articles.

1. Gastric-Outlet Obstruction Induced by Prostaglandin Therapy in Neonates

2. Cortical hyperostosis following long-term administration of prostaglandin E1 in infants with cyanotic congenital heart disease

3. Long-term low-dose prostaglandin E1 administration

4. Periosteal changes secondary to prostaglandin administration

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1. Associations between osteoporosis and drug exposure: A post-marketing study of the World Health Organization pharmacovigilance database (VigiBase®);Bone;2021-12

2. Effects of treatment with a bone-targeted prostaglandin E2 receptor 4 agonist C3 (Mes-1007) in a mouse model of severe osteogenesis imperfecta;Bone;2021-04

3. Prostaglandins and bone metabolism;Principles of Bone Biology;2020

4. Prostaglandins and Bone;Bone Regulators and Osteoporosis Therapy;2019

5. Prostaglandin E1 for maintaining ductal patency in neonates with ductal-dependent cardiac lesions;Cochrane Database of Systematic Reviews;2018-02-27

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