Protective effects of liquiritin on polycystic ovary syndrome through modulating ovarian granulosa cell proliferation and apoptosis via miR-206/PI3K/AKT pathway

Author:

Cui Xuan,Zhou Shisan,Lin YongtaoORCID

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Clinical Biochemistry,Biomedical Engineering,Bioengineering,Biotechnology

Reference34 articles.

1. Bachelot A (2016) Polycystic ovarian syndrome: clinical and biological diagnosis. Ann Biol Clin (paris) 74:661–667

2. Bao D, Li M, Zhou D, Zhuang C, Ge Z, Wei Q, Zhang L (2021) miR-130b-3p is high-expressed in polycystic ovarian syndrome and promotes granulosa cell proliferation by targeting SMAD4. J Steroid Biochem Mol Biol 209:105844

3. Bogari NM (2020) Genetic construction between polycystic ovarian syndrome and type 2 diabetes. Saudi J Biol Sci 27:2539–2543

4. Chen T, Chen J, Zhao X, Zhou J, Sheng Q, Zhu L, Lv Z (2021) betaKlotho, a direct target of miR-206, contributes to the growth of hepatoblastoma through augmenting PI3K/Akt/mTOR signaling. Am J Cancer Res 11:1982–2004

5. Du L, Huang GH, Mou KJ, Xiang Y, Tang JH, Xu W, Xia SL, Zhao JN, Lv SQ (2018) MiR-206 is down-regulated and suppresses cell proliferation by targeting FOXP1 in brain gliomas. Int J Clin Exp Pathol 11:3405–3415

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