Unilateral Moyamoya Disease: A Distinct Entity?
Author:
Publisher
Springer Singapore
Link
http://link.springer.com/content/pdf/10.1007/978-981-33-6404-2_3
Reference36 articles.
1. Mineharu Y, Takenaka K, Yamakawa H, Inoue K, Ikeda H, Kikuta K-I, et al. Inheritance pattern of familial moyamoya disease: autosomal dominant mode and genomic imprinting. J Neurol Neurosurg Psychiatry [Internet]. 2006 [cited 2010 Aug 31];77(August):1025–1029. Available from: http://jnnp.bmj.com/content/77/9/1025.full
2. Zhang Q, Wang R, Liu Y, Zhang Y, Wang S, Cao Y, et al. Clinical features and long-term outcomes of unilateral Moyamoya disease. World Neurosurg. 2016;96:474–82. https://doi.org/10.1016/j.wneu.2016.09.018.
3. Guidelines for diagnosis and treatment of moyamoya disease (Spontaneous Occlusion of the Circle of Willis). Neurol Med Chir [Internet]. 2012 Jan [cited 2013 Feb 18];52(5):245–266. Available from http://www.ncbi.nlm.nih.gov/pubmed/22870528
4. Tominaga T, Suzuki N, Miyamoto S, Koizumi A, Kuroda S, Takahashi JC, et al. Recommendations for the management of moyamoya disease: a statement from research committee on spontaneous occlusion of the circle of Willis (Moyamoya disease) [2nd edition]. Surg Cereb Stroke. 2018;46:1–24.
5. Morimoto T, Mineharu Y, Kobayashi H, Harada KH, Funaki T, Takagi Y, et al. Significant association of the RNF213 p.R4810K polymorphism with quasi-moyamoya disease. J Stroke Cerebrovasc Dis. 2016;25(11):2632–6. https://doi.org/10.1016/j.jstrokecerebrovasdis.2016.07.004.
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